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The asymmetric cell division cycle of Caulobacter crescentus is orchestrated by an elaborate gene-protein regulatory network, centered on three major control proteins, DnaA, GcrA and CtrA. The regulatory network is cast into a quantitative computational model to investigate in a systematic fashion how these three proteins control the relevant genetic, biochemical and physiological properties of proliferating bacteria. Different controls for both swarmer and stalked cell cycles are representeddoi:10.1371/journal.pcbi.1000463 pmid:19680425 pmcid:PMC2714070 fatcat:igeondrq4vazjixnvk3vwqnpmq