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Ovarian cancer (OC) represents the leading cause of gynecological cancer deaths in women. It has a high variety of histological origins, molecular pathways and genetic mutations (BRCA1 and BRCA2 somatic/germinal mutations, Lynch genes, RADS1C, RADS1D etc.) that play a key role in treatment response and prognosis (for example, clear cell carcinoma is less responsive to standard therapy). An important percentage of patients (80%) are diagnosed with metastatic disease, due to the lack of specificdoi:10.52701/monc.2020.v1i1.10 fatcat:iieaw4keubcv7d2vsz3muowfw4