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ВЛИЯНИЕ ПРЕНАТАЛЬНОГО СТРЕССА НА ОКИСЛИТЕЛЬНЫЕ МОДИФИКАЦИИ БЕЛКОВ И АКТИВНОСТЬ СУПЕРОКСИДДИСМУТАЗЫ В МОЗГЕ И ПЛАЗМЕ КРОВИ САМЦОВ КРЫС В МОДЕЛИ ПОСТТРАВМАТИЧЕСКОГО СТРЕССОВОГО РАССТРОЙСТВА
2018
Российский физиологический журнал им И М Сеченова
2 Pavlov Institute of Physiology of the RAS, St. Petersburg, Russia. Biochemical changes during development of posttraumatic stress disorder (PTSD) after prenatal stress were investigated in prenatally stressed male rats. Stress factors activate hypothalamus-pituitary-adrenal (HPA) axis thus increasing penetration of stress hormones into the fetus. It can disturb fetal HPA axis and lead to increased risk of stress-related disorders like PTSD. Our previous works showed that prenatally stressed
doi:10.1134/s0869813918110092
fatcat:ldezpnj7prclro2eg2jepzglwu