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Presence of Alternatively Spliced-Estrogen Receptor mRNA Variants in Normal Human Uterine Endometrium and Endometrial Cancer
Presence of alternatively spliced-estrogen receptor (ER) mRNA variants has been revealed in the breast cancer tissues. The ER variants transcribed from these mRNA variants were supposed to cause changes in the estrogen responsiveness of breast cancer. Although uterine endometrial cancer also has an estrogen-dependent profile, these ER mRNA variants have not yet been reported in the tumor. In the present study, we attempted to detect the exon 7 deletion-(del.7-) and exon 5 deletion (del.5) ERdoi:10.1507/endocrj.42.289 pmid:7542977 fatcat:boqg4tkvqfeq5kdtnm454ajadi