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Contribution of Single Nucleotide Polymorphisms withinFCRL3andMAP3K7IP2to the Pathogenesis of Graves' Disease
2006
Journal of Clinical Endocrinology and Metabolism
Context: Recently six DNA variants, two of which (M55V and 001Msp) are present in nuclear factor-B inhibitors SUMO-4 and MAP3K7IP2 and four of which (fcrl3_3, fcrl3_4, fcrl3_5, and fcrl3_6) modulate nuclear factor-B binding and production of the B cell surface molecule FCRL3, have been reported to be associated with a number of autoimmune diseases. Objective: The aim of this study was to investigate the association of these polymorphisms with disease in a large UK Caucasian Graves' disease (GD)
doi:10.1210/jc.2005-1634
pmid:16384851
fatcat:4qqhsthruvdync5nau3ujkdbpa