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Monocyte class switch and hyperinflammation characterise severe COVID-19 in type 2 diabetes
[article]
2020
medRxiv
pre-print
Early in the COVID-19 pandemic type 2 diabetes (T2D) was marked as a risk factor of severe disease and mortality. Inflammation is central to the aetiology of both conditions where variations in immune responses have the potential to mitigate or aggravate disease course. Identifying at risk groups based on immuno-inflammatory signatures is valuable in directing personalised care and developing potential targets for precision therapy. Methods: This study characterised immunophenotypic variation
doi:10.1101/2020.06.02.20119909
fatcat:2gbcux6uq5gkdhtqvep4iiogn4