A copy of this work was available on the public web and has been preserved in the Wayback Machine. The capture dates from 2017; you can also visit the original URL.
The file type is
Although the underlying mechanisms by which Kawasaki disease (KD) triggers vasculitis remain unclear, certain stimuli that release inflammatory substances trigger vasculitis during its acute phase, and may continue to cause vascular deterioration in subsequent phases of this disease. Cellular systems responsible for the generation and scavenging of reactive oxygen species (ROS) are clearly involved in the pathogenesis of KD vasculitis. In this manuscript, we discuss studies that illuminate thedoi:10.15344/2455-2364/2015/103 fatcat:t2zfq2gcmbdibcc5b35dlygspu