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Very recent studies hold promise to reveal the role of stromal interaction molecule 1 (STIM1) in non-storeoperated Ca 2+ entry. Here we showed that in contrast to cytoplasmic membrane redistribution as previously noted, human umbilical vein endothelial STIM1 with a T-to-C nucleotide transition resulting in an amino acid substitution of leucine by proline in the signal peptide sequence translocated to perinuclear membrane upon intracellular Ca 2+ depletion, amplified nucleoplasmic Ca 2+doi:10.1016/j.bbamcr.2016.01.007 pmid:26775216 fatcat:r5kucaraqnekrhqhb3alhfn5i4