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The influence of cognitive reserve on inter-individual variability in resting-state cerebral metabolism in normal aging
2012
Alzheimer's & Dementia
atrophy and intracellular accumulations of tau, With the exception of a small number of familial forms the causes for FTLD are unknown. With our plasma proteomics approach we identified novel disease pathways that may open possibilities for treatment and diagnostics. Methods: We employed medium scale plasma proteomics, measuring more than 600 soluble cellular communication factors in plasma from control (n ¼ 83) and FTLD (n ¼ 92) patients with semantic dementia, as well as from FTLD patients
doi:10.1016/j.jalz.2012.05.193
fatcat:yjnnxqzzwncd3ob5dzwpct3g5u