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Deletions in mitochondrial DNA occur from oxidative stress generated during physiological processes. Such deletions have been associated with ageing, cancer, and neurodegeneration. In this study, the outcome of mtDNA deletions has been investigated to justify the physiological effect of such mtDNA deletion. For this, five established and two predicted age-related murine mtDNA deletions were analyzed for fusion peptide. Three out of the five established deletions and all the predicted deletionsdoi:10.18034/ei.v6i1.172 fatcat:och7egbnbzgspckh2rqttxev3m