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REEP5 (Receptor Accessory Protein 5) Acts as a Sarcoplasmic Reticulum Membrane Sculptor to Modulate Cardiac Function
2018
Journal of the American Heart Association : Cardiovascular and Cerebrovascular Disease
Heart failure is a complex syndrome characterized by cardiac contractile impairment with high mortality. Defective intracellular Ca2+ homeostasis is the central cause under this scenario and tightly links to ultrastructural rearrangements of sarcolemmal transverse tubules and the sarcoplasmic reticulum (SR); however, the modulators of the SR architecture remain unknown. The SR has been thought to be a specialized endoplasmic reticulum membrane system. Receptor accessory proteins
doi:10.1161/jaha.117.007205
pmid:29431104
pmcid:PMC5850239
fatcat:jwix6mfvqrdblnw45j7hpgzutu