A copy of this work was available on the public web and has been preserved in the Wayback Machine. The capture dates from 2019; you can also visit the original URL.
The file type is application/pdf
.
Retinotopically defined primary visual cortex in Williams syndrome
2009
Brain
Williams syndrome, caused by a hemizygous microdeletion on chromosome 7q11.23, is characterized by severe impairment in visuospatial construction. To examine potential contributions of early visual processing to this cognitive problem, we functionally mapped the size and neuroanatomical variability of primary visual cortex (V1) in high-functioning adults with Williams syndrome and age-and IQ-matched control participants from the general population by using fMRI-based retinotopic mapping and
doi:10.1093/brain/awn362
pmid:19255058
pmcid:PMC2724925
fatcat:f465xvxx2vhf5b7nhjubl2gyfq