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The purpose of the current study is to investigate the effect of opioid-independent, heterologous activation of protein kinase C (PKC) on the responsiveness of opioid receptor and the underlying molecular mechanisms. Our result showed that removing the C terminus of ␦ opioid receptor (DOR) containing six Ser/Thr residues abolished both DPDPE-and phorbol 12-myristate 13acetate (PMA)-induced DOR phosphorylation.doi:10.1074/jbc.m006187200 pmid:11085981 fatcat:l7pc35txtrdyfjb2aeajbvrdyi