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Understanding potential mechanisms of harm: the drivers of electronic cigarette-induced changes in alveolar macrophages, neutrophils and lung epithelial cells
2021
American Journal of Physiology - Lung cellular and Molecular Physiology
Electronic (e-) cigarettes are growing in popularity despite uncertainties regarding their long-term health implications. The link between cigarette smoking and initiation of chronic lung disease took decades to unpick so in vitro studies mimicking e-cigarette exposure aim to detect early indicators of harm. In response to e-cigarette exposure, alveolar macrophages adopt a pro-inflammatory phenotype of increased secretion of proinflammatory cytokines, reduction in phagocytosis and efferocytosis
doi:10.1152/ajplung.00081.2021
pmid:34009037
fatcat:xmrggxxezff7bo23ukhif23gdi