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Sodium valproate, one of the most widely used antiepileptic drugs, has been noted to induce nephrotoxicity through elevation of oxidative stress. Carvedilol, a non-specific beta-adrenergic blocker, and rosuvastatin, an anti-hyperlipidemic, possess antioxidant characteristics. This study was planned to estimate the promising defensive impacts of Carvedilol and/or rosuvastatin against valproate-induced nephrotoxicity. It was revealed that sodium valproate (SVP) markedly boosted serum creatininedoi:10.21608/aijpms.2021.206679 fatcat:my7ksg54w5garinrvnwfqteoci