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1. ABSTRACT There has been much research into autism or autistic spectrum disorder (ASD) and room for considerable conjecture regarding the etiology of these disorders remain. ASD is marked by a complex interaction between environmental factors and genetic predisposition, including epistasis. This manuscript argues that changes in oxidative metabolism, thiamine homeostasis, heavy metal deposition and cellular immunity have a role in the etiopathogenesis of autism and ASD. Recent evidence fordoi:10.2741/e730 fatcat:fkilvopekveu3caque356t3h4y