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Changes in indoleacetic acid production and chromosome length polymorphism in clofibric acid resistant mutants of Taphrina wiesneri and Taphrina deformans
1990
Japanese Journal of Phytopathology
Taphrina wiesneri and T. deformans, causal agents of plant hyperplastic diseases, produce indoleacetic acid (IAA) via indolepyruvic acid and indoleacetaldehyde from L-tryptophan as intermediates. They also convert indoleacetonitrile into IAA. We have isolated Iaa-mutants of these Taphrina spp, by the treatment with clofibric acid. These mutants are Pur-(deficient in purine biosynthesis) and exhibit unique morphology including the smaller conidium size. They can no longer accumulate IAA in
doi:10.3186/jjphytopath.56.651
fatcat:nnntfrnhfrdwtgbpmjszrup5sa