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The main aim of this study was to estimate the relative influence of genes and environment on fasting insulin levels, which were considered a proxy of insulin resistance. Possible sex differences in genetic and environmental influences, and the origin of the covariance between fasting insulin and glucose were investigated. Subjects were 209 pairs of middle-aged twins, divided into 5 sex-by-zygosity groups. A general bivariate model and a reciprocal causation model including fasting insulin anddoi:10.1002/(sici)1098-2272(1999)16:4<426::aid-gepi8>3.0.co;2-b pmid:10207722 fatcat:wieejqbvrraxbm57oq442toq6a