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Background&Aims: Intestinal gluconeogenesis (IGN), gastric bypass (GBP) and gut microbiota positively regulate glucose homeostasis and diet-induced dysmetabolism. GBP modulates gut microbiota but whether IGN intensity could shape it has not been investigated. Methods: To this aim, we studied gut microbiota and microbiome in wild-type and IGN-deficient mice which underwent GBP and were fed on either a normal chow (NC) or a high-fat/high-sucrose (HFHS) diet. We also studied fecal and urinedoi:10.21203/rs.3.rs-892275/v1 fatcat:77pkkqdicbcrblrwv2vodws7iq