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Cellular models of cardiac hypertrophy and cardiac failure suggest that haemodynamic stresses lead to increased rates of cardiac myocyte apoptosis and ®brosis. Over the last 15 years, it has been become evident that the dramatically ampli®ed exposure of patients with renal insuf®ciency to haemodynamic stress leads to maladaptive vascular and ventricular adaptations. Anaemia and hypertension are remediable haemodynamic stresses consistently associated with left ventricular enlargement indoi:10.1093/ndt/16.suppl_1.102 pmid:11369834 fatcat:xsxefpxeljfwxp7g4jkfftdg2m