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The influenza A virus (IAV) causes a respiratory tract infection with approximately 10% of the population infected by the virus each year. Severe IAV infection is characterized by excessive inflammation and tissue pathology in the lungs. Platelet and neutrophil recruitment to the lung are involved in the pathogenesis of IAV, but the specific mechanisms involved have not been clarified. Using confocal intravital microscopy in a mouse model of IAV infection, we observed profound neutrophildoi:10.3389/fimmu.2021.772859 pmid:34858432 pmcid:PMC8632260 fatcat:fxmklfoh2zdtdoexkvmtdgom3e