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Prenatal bisphenol A exposure is associated with medial amygdala neuron hyperresponsiveness to predator odor in rats
Journal of Toxicological Sciences
Perinatal exposure to bisphenol A (BPA) causes several alterations in brain function and behavior. In previous studies, we showed that prenatal treatment with low-level BPA impaired gender-specific behavior, enhanced depression-like behavior, and augmented behavioral responses to predator odor in rats. On this premise, we hypothesized that BPA-treated rats were more susceptible to predator odor stress. To test the potential neural mechanism underlying this effect, we conducted andoi:10.2131/jts.43.531 pmid:30185693 fatcat:taausm7ymfeqtc4lrenoehmx6q