A copy of this work was available on the public web and has been preserved in the Wayback Machine. The capture dates from 2019; you can also visit the original URL.
The file type is
We appreciate the comments of Leiro et al. on the role of the combined glutathione-S-transferase M1 (GSTM1) and glutathione-S-transferase T1 (GSTT1) null genotypes in determining the susceptibility to developing idiosyncratic drug-induced liver injury (DILI) as a general mechanism that occurs regardless of the type of drug involved. 1 These enzymes are predominantly expressed in the liver, and apart from being involved in drug metabolism, they modulate the byproducts of oxidative stress, an important common This letter was written on behalfdoi:10.1002/hep.22922 fatcat:jrvwtftvabbbri6mpwpxh2lx54