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AXONAL DEGENERATION IN THE CENTRAL NERVOUS SYSTEM OF GRACILE AXONAL DYSTROPHY (GAD) MICE PROGRESSES LIKE IN HUMAN SPINOCEREBELLAR ATAXIAS
We examined neuropathologically the central nervous system (CNS) of gracile axonal dystrophy (GAD) mice from C57BL/6-gad strain, at 115-135 days (critical stage) and 238-272 days of age (terminal stage). Axonal degeneration and spheroid formation (axonal dystrophy) were observed in the gracile tract, dorsal spinocerebellar tract, and spinal trigeminal nucleus at the critical stage. At the terminal stage, their distribution spread to the corticospinal (pyramidal) tract, cuneate tract, spinaldoi:10.2220/biomedres.12.143 fatcat:2tkmwfc6onh7rdy7qsmmusasgi