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Common forms of metabolic and cardiovascular diseases involve the interplay of numerous genes as well as important environmental factors. Traditional biochemical and genetic approaches generally attempt to dissect these diseases one gene at a time, for example, by analysis of Mendelian forms or genetically engineered experimental organisms. But, it is also important to understand how the genes interact with each other and the environment, and how these interactions change in disease states.doi:10.1194/jlr.r800067-jlr200 pmid:19033210 pmcid:PMC2674684 fatcat:4kwq45wpgbemlkytavad3jrbaq