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Heart attacks caused by occlusion of coronary arteries are often treated by mechanical or enzymatic removal of the occlusion and reperfusion of the ischemic heart. It is now recognized that reperfusion per se contributes to myocardial damage, and there is a great interest in identifying the molecular basis of this damage. We recently showed that inhibiting protein kinase C␦ (PKC␦) protects the heart from ischemia and reperfusion-induced damage. Here, we demonstrate that PKC␦ activity anddoi:10.1074/jbc.m405071200 pmid:15339931 fatcat:ttah7volufhwpo6z26wer3olqq