A copy of this work was available on the public web and has been preserved in the Wayback Machine. The capture dates from 2018; you can also visit the original URL.
The file type is application/pdf
.
Essential Role of Gap Junctions in NO- and Prostanoid-Independent Relaxations Evoked by Acetylcholine in Rabbit Intracerebral Arteries
2003
Stroke
and Purpose-Direct intercellular communication via gap junctions may play a central role in endothelium-dependent relaxations that are mediated by a conducted hyperpolarization and do not involve the synthesis of NO and prostanoids. In the present study, inhibitory peptides homologous to the Gap27 domain of the second extracellular loop of connexin37/connexin43 and connexin40, designated as 37,43 Gap27 and 40 Gap27, respectively, were used to evaluate the role of this mechanism in intracerebral
doi:10.1161/01.str.0000054158.72610.ec
pmid:12574573
fatcat:44z46aqnr5ctvdzptasklmedwy