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Protein kinase M zeta (PKMζ) is assumed to be actively involved in retainig long-term potentiation. The goal of this study was to investigate the role of PKMζ in basolateral amygdala (BLA) upon acquisition, consolidation, retention and retrieval of memory using a specific inhibitor of PKMζ. Methods: Sixty male wistar rats underwent stereotaxic surgery and were cannulated bilaterally at the BLA nucleus. Then animals were divided into 4 groups of receiving BLA microinjection of zeta inhibitoryfatcat:rwzvnwvrifhcvfowhtywvrsola