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AbstractMalaria begins when mosquito-borne Plasmodium sporozoites invade hepatocytes and usurp host pathways to support the differentiation and multiplication of erythrocyte-infective, merozoite progeny. All Plasmodium species encode an orthologue of the innate cytokine, Macrophage Migration Inhibitory Factor (MIF), which functions in mammalian biology to regulate innate responses. Using a genetically-targeted strain of Plasmodium berghei, we demonstrate that the Plasmodium MIF orthologue,doi:10.1101/2021.02.14.430970 fatcat:zo7pflqgnneldlppuec6ey566e