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Differential Expression of Desmocollin2, Desmoglein2, and Plakophilin2 in the Progression of Esophagitis to Esophageal Adenocarcinoma
BackgroundDesmosomes play a key role in intercellular adhesive, but also contribute to tumorigenesis. This study aimed to examine the differential expression of desmocollin2 (DSC2), desmoglein2 (DSG2), and plakophilin2 (PKP2) in the progression of reflux esophagitis to esophageal adenocarcinoma (EAC) in the rat model of reflux disease established by esophagogastroduodenal anastomosis (EGDA).MethodsEGDA was performed on rats to induce gastroesophageal reflux leading to the development of EAC.doi:10.21203/rs.3.rs-154489/v1 fatcat:btihbltlejea7dc4cfkg6puovy