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Amyloid diseases are global epidemics with profound health, social and economic implications and yet remain without a cure. This dire situation calls for research into the origin and pathological manifestations of amyloidosis to stimulate continued development of new therapeutics. In basic science and engineering, the cross-b architecture has been a constant thread underlying the structural characteristics of pathological and functional amyloids, and realizing that amyloid structures can bedoi:10.3929/ethz-b-000427996 fatcat:dpyr4nxdhbaejfqcvsvygrjkou