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Dual Epigenetic Regulation of ERα36 Expression in Breast Cancer Cells
2019
International Journal of Molecular Sciences
Breast cancer remains the major cause of cancer-induced morbidity and mortality in women. Among the different molecular subtypes, luminal tumors yet considered of good prognosis often develop acquired resistance to endocrine therapy. Recently, misregulation of ERα36 was reported to play a crucial role in this process. High expression of this ERα isoform was associated to preneoplastic phenotype in mammary epithelial cells, disease progression, and enhanced resistance to therapeutic agents in
doi:10.3390/ijms20112637
fatcat:wwdbyzrc6fer7if6vfeuqerniu