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Differentially regulated splice variants and systems biology analysis of Kaposi's sarcoma-associated herpesvirus-infected lymphatic endothelial cells
2011
Nucleic Acids Research
Alternative RNA splicing greatly increases proteome diversity, and the possibility of studying genomewide alternative splicing (AS) events becomes available with the advent of high-throughput genomics tools devoted to this issue. Kaposi's sarcoma associated herpesvirus (KSHV) is the etiological agent of KS, a tumor of lymphatic endothelial cell (LEC) lineage, but little is known about the AS variations induced by KSHV. We analyzed KSHV-controlled AS using high-density microarrays capable of
doi:10.1093/nar/gkr405
pmid:21646333
pmcid:PMC3167639
fatcat:klocnbchaffkfg7rwllab7m6pm