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Stem Cell Reports
Hormones drive mammary development and function and play critical roles in breast cancer. Epidemiologic studies link prolactin (PRL) to increased risk for aggressive cancers that express estrogen receptor a (ERa). However, in contrast to ovarian steroids, PRL actions on the mammary gland outside of pregnancy are poorly understood. We employed the transgenic NRL-PRL model to examine the effects of PRL alone and with defined estrogen/progesterone exposure on stem/progenitor activity anddoi:10.1016/j.stemcr.2017.08.011 pmid:28919264 pmcid:PMC5639259 fatcat:i7fgqciciffvrgesoimlks62su