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Dimethyl Disulfide Exerts Insecticidal Neurotoxicity Through Mitochondrial Dysfunction and Activation of Insect KATP Channels
2003
Journal of Neurophysiology
Dimethyl disulfide exerts insecticidal neurotoxicity through mitochondrial dysfunction and activation of insect K ATP channels. J Neurophysiol 90: 259 -270, 2003; 10.1152/jn.01096.2002. The plant-derived insecticides have introduced a new concept in insecticide research. In response to insect attacks, some plants can release volatile sulfur compounds such as dimethyl disulfide (DMDS) in the atmosphere, which are lethal for the generalist insects. We demonstrate that DMDS induced an uncommon
doi:10.1152/jn.01096.2002
pmid:12843310
fatcat:s33w4vgbhjcwni225wvyseexqe