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Amyloid-β (Aβ) peptides are proteins associated with Alzheimer's disease (AD), because the extracellular Aβ deposits are the main cause of this disorder. The aggregation of Aβ has been shown to depend on the interactions with metal ions, such as copper, zinc, aluminum or iron. The N-terminal sequence of Aβ or Aβ (1-40) peptides, namely Aβ (1-16) peptide fragment, is considered the metal binding site involved in AD neurodegeneration and amyloidogenesis. Therefore, we have investigated differentdoi:10.37358/rc.19.9.7547 fatcat:bnx6qbxd7zczhe4gwdcjcibbye