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While a general consensus exists about the biochemical mechanism that underlies the initiation step of SM contraction,4 the subsequent phase concerning stress maintenance appears less clear. Active stress in a variety of intact SM preparations can be maintained independent of phosphorylation.811-15 Thus, phosphorylation of SM myosin correlates better with the shortening velocity of SM tissue.'6 It has been suggested that a second Ca'+-dependent regulatory mechanism could be involved in thedoi:10.1161/01.res.68.5.1349 pmid:2018995 fatcat:vmrqh2ai7bhsxbinr7ip7e6oqq