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Human immediate early response 2 (IER2) has been reported to function as a potential transcriptional factor or transcriptional co-activator and seems to play a pivotal role in tumor cell motility and metastasis, however, its role and underlying mechanisms in hepatocellular carcinoma (HCC) remain unknown. Herein, we demonstrated that overexpression of IER2 in HCC cells increased cell adhesion to fibronectin, migration and invasion, whereas knockdown of IER2 displayed the opposite effects. Indoi:10.3892/or.2016.5215 pmid:27840969 fatcat:7ssrzqdbwjfh5k6wndx53ozslq