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Patients in high-risk groups continue to transmit the hepatitis C virus (HCV) and frequently experience reinfections. Since little is known regarding the immune response to HCV during reinfection, we compared primary and consecutive acute HCV infections in patients with an HIV infection, and focused on the cytokine bridging innate to adaptive immunity. We observed that the serum levels of IL-12p40, MIP-1b, MIG and IP-10 increased during primary acute HCV infection, but not during subsequentdoi:10.1099/jgv.0.000768 pmid:28597817 fatcat:eu5zgrarafhz5fjyqcuombnxnq