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Role of the Central Amygdala in Modulating the Pituitary-Adrenocortical and Clinical Responses in Experimental Herpes Simplex Virus-1 Encephalitis
tor hyperactivity and aggressive behavior. These responses were also attenuated in rats with cAMG lesions. The cAMG plays an important role in mediating the neuroendocrine, febrile and behavioral responses to HSV-1 infection. Abstract The amygdala is known to regulate neuroendocrine and behavioral responses to a variety of stimuli. Herpes simplex virus-1 (HSV-1) is the common cause of viral encephalitis, manifested by hypothalamic-pituitary-adrenal (HPA) axis activation, fever, hypermotordoi:10.1159/000087924 pmid:16131813 fatcat:ghwqucmkwzdi3gnomezzpdiwge