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Aims/hypothesis Genome-wide association studies (GWAS) have discovered many risk variants for type 2 diabetes. However, estimates of the contributions of risk variants to type 2 diabetes predisposition are often based on highly selected case-control samples, and reliable estimates of populationlevel effect sizes are missing, especially in non-European populations. Methods The individual and cumulative effects of 59 established type 2 diabetes risk loci were measured in a population-based Chinadoi:10.1007/s00125-016-3920-9 pmid:27053236 pmcid:PMC4901105 fatcat:havk74cgzzhyxnoyt5vnhgsfqu