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I ONIC FLUX across the plasma membrane through voltage-dependent and -independent ionic channels are important in the regulation of many cellular functions, including hormone release. In mammals, LH release is stimulated by the hypothalamic decapeptide, mammalian GnRH (1). Activation of GnRH-receptors in the plasma membrane results in the oscillatory release of intracellular Ca*+ from InsI',-sensitive stores, which leads to the generation of periodic membrane hyperpolarizations followed by onedoi:10.1210/en.137.7.2859 pmid:8770907 fatcat:bcnrjdts5faoxpyhvshxdwc65i