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These authors contributed equally to this work Purpose: Myocardial delivery of magnetic iron oxide nanoparticles (MNPs) might produce iron overload-induced myocardial injury, and the oxidative stress was regarded as the main mechanism. Therefore, we speculated antioxidant modification might be a reasonable strategy to mitigate the toxicity of MNPs. Methods and results: Antioxidant N-acetylcysteine (NAC) was loaded into magnetic mesoporous silica coated Fe 3 O 4 nanoparticles. Neonatal ratdoi:10.2147/ijn.s209820 fatcat:ewbaggpbhvfzfmd6e3x23cqriy