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A molecular mechanism for membrane chaperoning by a late embryogenesis abundant protein
[article]
2022
bioRxiv
pre-print
Environmental stress can result in substantial damage to proteins, membranes, and genetic material, impacting organismal survival. Stress tolerance can be conferred by intrinsically disordered proteins (IDPs) that lack stable tertiary structure. IDPs from the large family of late embryogenesis abundant (LEA) proteins confer a fitness advantage when heterologously expressed. Such protection suggests a general molecular function leading to stress tolerance, although the mechanisms remain unclear.
doi:10.1101/2022.07.29.502075
fatcat:pygg42xhhzhedh547bctye3lgi