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129X1/SvJ Mouse Strain Has a Novel Defect in Inflammatory Cell Recruitment
Journal of Immunology
Vitamin D-binding protein (DBP) has been reported to contribute to innate immunity. To verify prior in vitro and cell-based observations supporting this role, we assessed the ability of a recently developed DBP-null mouse line to recruit neutrophils and macrophages to a site of chemical inflammation. The interrupted DBP allele had been generated by homologous recombination in 129X1/SvJ embryonic stem cells and these cells were subsequently used to generate a line of DBP ؊/؊ (null) mice. Initialdoi:10.4049/jimmunol.168.2.869 pmid:11777984 fatcat:mbvljm52s5dh3hfw5upvm4gtzq