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Molecular interactions in the cell are not bound by discrete cellular pathways; there is significant communication between the pathways leading to tightly regulated processes. The current research will examine if Wnt and NFκB pathway component interplay can stimulate endothelial to mesenchymal transition (EMT) via the cell-junction component β-catenin (Ctnn-B). The classical Wnt pathway utilizes Ctnn-B as a signaling molecule; specifically, under activated canonical Wnt signaling to inducedoi:10.13016/m25717q5q fatcat:cljop3kgy5aytlyt3zzfajqf6e