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NF-κB pathway link with ER stress-induced autophagy and apoptosis in cervical tumor cells
2017
Cell Death Discovery
Targeting endoplasmic reticulum (ER) stress is being investigated for its anticancer effect in various cancers, including cervical cancer. However, the molecular pathways whereby ER stress mediates cell death remain to be fully elucidated. In this study, we confirmed that ER stress triggered by compounds such as brefeldin A (BFA), tunicamycin (TM), and thapsigargin (TG) leads to the induction of the unfolded protein response (UPR) in cervical cancer cell lines, which is characterized by
doi:10.1038/cddiscovery.2017.59
pmid:28904818
pmcid:PMC5592653
fatcat:xyf7olm7xncylax6jslua4uzra