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The involvement of increased brain tissue CO2 tension in acetazolamide-induced brain acidosis was in vestigated by comparing the brain pH response to acetazolamide with that to hypercapnia. CO2 and pH sensors were placed bilaterally into cerebral white matter to 15 mm depth in cats. Group I cats (n = 9) breathed spontaneously, and in situ brain tissue PCO2 and pH (PCO2 and pHb) were measured after in travenous acetazolamide administration (20 mg/kg). Group 2 cats (n = 9) were paralyzed anddoi:10.2176/nmc.37.313 pmid:9136554 fatcat:pi6nuqb755ccvd646as6yfcvmu