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While the exact ammonia-induced pathogenic mechanisms in hepatic encephalopathy (HE) are not completely understood, an increasing number of reports have lately been involved in studying the possible relevance of oxidative stress status in HE. Here we evaluated the levels of some oxidative stress markers (two main antioxidant enzymes: superoxide dismutase-SOD and glutathione peroxidase-GPX, as well as a lipid peroxidation marker: malondialdehyde-MDA) in thirty-six cirrhotic patients with overtdoi:10.2298/abs1303245i fatcat:rtuv3ee7e5gpbj2q3oq2sraypa