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The hypophosphatemic (Hyp) mouse is a useful model for studying certain genetic defects in renal phosphate (Pi) handling in humans. Sodium-dependent phosphate transport activity was measured in brush border membrane vesicle (BBMV) prepared from renal cortex and in Xenopus oocytes that had been injected with renal poly(A)+RNA. Pi transport activity in Hyp mice was significantly reduced compared with the control. Northern blot analysis revealed that the level of NaPi-7 mRNA was reduced in Hypdoi:10.1297/cpe.5.89 fatcat:enfdcosxdbaadd5uwluw5egyde